Publication: Health Tips Weekly
Some cancer drugs bad for heart patients | |
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HEALTH TIPS WEEKLY - Thursday, June 26, 2008
"News That Keeps You Healthy"
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'Grid computing' may help fight cancer
AUSTIN, Texas, -- A U.S. biomedical engineering professor is
using the concept called "grid computing" to allow people to
donate their idle computer time to fight cancer. University
of Texas at Austin Assistant Professor Muhammad Zaman has
introduced a grid program called Cellular Environment in
Living Systems at home -- or CELS@Home. It allows Internet
users worldwide to contribute their idle computer time,
creating a "virtual" supercomputer to solve a difficult
problem. Zaman's "grid computing" program is helping scien-
tists understand the principles of cell migration and cancer
cell metastasis. He said the program has already yielded
enough information in just two months for two scientific
journal articles. He said only a screensaver needs to be
downloaded -- at no cost to the user -- to contribute to
the CELS@Home effort. A computational program then runs
whenever the screensaver is activated, requiring no effort
on the part of the user to run the program or report the
computations. People interested in participating in the
program can obtain more information at ...
http://cels-at-home-dev.dyndns.org/cels/
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New stem cells can regenerate heart tissue
BOSTON,-- U.S. scientists say they've identified a new group
of stem cells that can create cardiomyocytes, or heart
muscle cells, to regenerate injured heart tissue. The find-
ing by researchers at Children's Hospital Boston follows
studies that found a specific stem cell or progenitor,
marked by expression of a gene called Nkx2-5 can form many
components of the heart, including heart muscle cells. In
the new finding, Children's Hospital scientists showed heart
muscle cells can also be derived from a third type of car-
diac progenitor located within the epicardium, or the
heart's surface, and identifiable through its expression of
a gene called Wt1. "There's a lot of interest in finding
places to obtain new cardiomyocytes, because in heart fail-
ure you lose cardiomyocytes, so the only way to reverse
heart failure is to make more of these cells," said
Dr. William Pu, who led the research. Although epicardial
cells are known to give rise to smooth muscle and endothel-
ial cells during coronary vessel formation, nobody previ-
ously thought epicardial cells might turn into cardiomyo-
cytes. The results were independently corroborated by res-
earchers from the University of California-San Diego. The
findings are reported online in the journal Nature.
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Some cancer drugs bad for heart patients
ST. LOUIS,-- U.S. medical scientists say a set of promising
new anti-cancer agents called hedgehog antagonists might
present a risk for people with heart disease. Researchers
at the Washington University School of Medicine in St. Louis
said the hedgehog antagonists interfere with a biochemical
process called hedgehog signaling that promotes growth in
some cancer cells. But the study showed interfering with
that biochemical process in mice with heart disease led to
further deterioration of cardiac function and ultimately
death. "This finding should serve as a warning that these
drugs might have adverse effects on the heart and that it
could be very important to monitor patients' cardiovascular
health when using this type of anti-cancer drug," said sen-
ior study author Dr. David Ornitz. He said the study is the
first demonstration that hedgehog signaling is essential to
maintain a healthy supply of blood vessels in adult heart
muscle. The research that included Dr. Kory Lavine appears
in the advance online issue of the Journal of Clinical
Investigation.
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Scientists ID new Alzheimer's disease gene
MANHASSET, N.Y.,-- U.S. medical scientists say they have
identified a gene that puts people at risk for developing
Alzheimer's disease. Philippe Marambaud of the Feinstein
Institute for Medical Research and Fabien Campagne of the
Weill Medical College at Cornell University said the calcium
channel modulator gene holds promise as a potentially new
way to treat or even prevent Alzheimer's disease. They said
they found the risk gene called CALHM1 strongly expressed in
the hippocampus -- a brain region necessary for learning and
memory. The researchers said CALHM1 leads to a partial loss
of function, which means less calcium gets into a cell,
thereby weakening signals normally regulated by calcium.
They determined one of those signals controls the levels of
amyloid peptides, the building blocks of the characteristic
disease plaques. Using DNA from deceased U.S. Alzheimer's
victims and age-matched controls, along with DNA samples
from patients in France, Italy and the United Kingdom, the
researchers ran tests on 3,404 samples. They discovered
people having the genetic variant had 1.5 times higher risk
of developing Alzheimer's. The study that included French
researcher Jean Charles Lambert is detailed in the journal
Cell.
--------------- Health Tip Video of the Week ---------------
Health Tip: Isophane Insulin - current rating 3.6
A pharmacist explains how Humalog works, why doctors prescribe
this medication to diabetics, and common side effects of the
drug.
Health Tip: Maximize Memory
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