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New airway bypass for emphysema studied | |
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HEALTH TIPS WEEKLY - Thursday, December 20, 2007
"News That Keeps You Healthy"
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FDA approves a new beta blocker
WASHINGTON, -- The U.S. Food and Drug Administration has
approved Bystolic, a beta blocker, for the treatment of
high blood pressure. Bystolic (nebivolol) is a new drug not
previously approved for use in the United States. Beta
blockers are a well-established class of medications that
reduce blood pressure by reducing the force with which the
heart pumps, the FDA said. Nearly 1-in-3 adults in the
United States suffers from hypertension, which can increase
the risks for stroke, heart failure, heart attack, kidney
failure, and death. The FDA said the safety and efficacy of
Bystolic was assessed during three randomized, double-blind,
multi-center, placebo-controlled clinical trials that ran
for up to three months. A fourth placebo-controlled clinical
trial demonstrated additional blood pressure-lowering eff-
ects when Bystolic was given with up to two other antihyper-
tensive medications in patients with inadequate blood press-
ure control. More than 2,000 people received Bystolic during
the trials, the FDA said, with the most common side effects
reported being headache, fatigue, dizziness and diarrhea.
Mylan Bertek Pharmaceuticals Inc. of Research Triangle Park,
N.C., is the sponsor of Bystolic. New York City-based Forest
Laboratories Inc. owns the rights for marketing of the drug.
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Breast cancer stem cells repressed in mice
COLD SPRING HARBOR, N.Y., -- U.S. scientists at the Cold
Spring Harbor Laboratory have isolated and repressed stem-
like cells in mouse breast cancer tissue. By manipulating
highly specific gene-regulating molecules called microRNAs,
the researchers said they were able to repress the cells
that are widely thought to give rise to cancer. "If certain
forms of breast cancer do indeed have their origin in way-
ward stem cells, as we believe to be the case, then it is
critical to find ways to selectively attack that tumor-
initiating population," said Professor Gregory Hannon, corr-
esponding author of the study. "We have shown that a micro-
RNA called let-7, whose expression has previously been ass-
ociated with tumor suppression, can be delivered to a sample
of breast-tissue cells, where it can help us to distinguish
stem-like tumor-initiating cells from other, more fully dev-
eloped cells in the sample. "Even more exciting, we found
that by expressing let-7 in the sample, we were able to
attack and essentially eliminate, very specifically, just
that subpopulation of potentially dangerous progenitor
cells," Hannon added.
MIT works toward engineered blood vessels
CAMBRIDGE, Mass., -- U.S. scientists have created a tech-
nique that induces cells to form parallel tube-like struc-
tures that might serve as tiny engineered blood vessels.
Massachusetts Institute of Technology researchers said they
found a way to control cell development by growing them on
a surface with nanoscale patterning. The scientists said
engineered blood vessels might one day be transplanted into
tissues such as the kidneys, liver, heart or any other
organs that require large amounts of vascular tissue, which
moves nutrients, gases and waste to and from cells. "We are
very excited about this work," said Professor Robert Langer,
an author of the study. "It provides a new way to create
nano-based systems with what we hope will provide a novel
way to someday engineer tissues in the human body."
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Bacteria might cause bladder re-infection
ST. LOUIS, -- A U.S. study suggested that intracellular
bacteria that commonly exist in women with bladder infect-
ions might contribute to the recurrence of such infections.
Researchers at the Washington University School of Medicine
in St. Louis and Washington University in Seattle noted it
was previously established in studies with mice that the E.-
coli bacterium avoids the immune system by invading cells
lining the bladder, then replicates and ultimately reinfects
the urinary tract. The scientists said the existence of IBCs
found in the new study suggests a similar cycle might also
occur in people and that longer treatment with antibiotics
that kill bacteria inside human cells might be necessary for
some patients.
New airway bypass for emphysema studied
SARASOTA, Fla., -- U.S. medical scientists are joining an
international team starting a study of a minimally invasive
airway bypass treatment for emphysema patients. Researchers
at Sarasota Memorial Health Care System in Sarasota, Fla.,
announced the start Monday of the Exhale Airway Stents for
Emphysema Trial. The multi-center clinical trial will ex-
plore a treatment that might offer a significant new, mini-
mally invasive option for those suffering with advanced
widespread emphysema. The study focuses on a procedure
called airway bypass that involves creating pathways in
the lung for trapped air to escape, thereby relieving emph-
ysema symptoms. "We are excited to be part of this study
because currently treatment options for the emphysema pat-
ients are very limited and many patients have a very poor
quality of life," said Dr. Kirk Voelker, principal investi-
gator of the study at Sarasota Memorial Hospital. "By creat-
ing new pathways for airflow with the airway bypass proc-
edure, we hope to reduce hyperinflation and improve lung
function. If patients can breathe easier it is likely to
improve their quality of life."
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Heart protein linked to muscular dystrophy
CHARLOTTESVILLE, Va., -- U.S. medical scientists have disc-
overed a well known heart protein, NKX2-5, is linked with
type-1 myotonic muscular dystrophy. Researchers at the Uni-
versity of Virginia Health System discovered that, in cases
of type 1 myotonic muscular dystrophy, or DM1, NKX2-5 is
overproduced in mice and people with DM1, yet it produces
the same kind of heart problems associated with a deficiency
of the protein. The scientists said DM1 is the most common
form of muscular dystrophy in adults and NKX2-5 is a bio-
marker for heart stem cells, as well as being very important
for the normal development of the heart. "Too little of it
causes major cardiac problems including slow and irregular
heartbeats," said Dr. Mani Mahadevan, a human genetics res-
earcher and a professor of pathology who led the study. Mah-
adevan said excessive NKX2-5 might explain why as many as
70 percent of individuals with DM1 develop heart problems
that cause their heartbeats to become slow and irregular,
often necessitating the need for pacemakers.
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